My watch list
my.chemeurope.com  
Login  

Loss-of-function mutations in sodium channel Nav1.7 cause anosmia

Loss-of-function mutations in sodium channel Nav1.7 cause anosmia

Nature 472, 7342 (2011). doi:10.1038/nature09975

Authors: Jan Weiss, Martina Pyrski, Eric Jacobi, Bernd Bufe, Vivienne Willnecker, Bernhard Schick, Philippe Zizzari, Samuel J. Gossage, Charles A. Greer, Trese Leinders-Zufall, C. Geoffrey Woods, John N. Wood & Frank Zufall

Loss of function of the gene SCN9A, encoding the voltage-gated sodium channel Nav1.7, causes a congenital inability to experience pain in humans. Here we show that Nav1.7 is not only necessary for pain sensation but is also an essential requirement

Authors:   Jan Weiss; Martina Pyrski; Eric Jacobi; Bernd Bufe; Vivienne Willnecker; Bernhard Schick; Philippe Zizzari; Samuel J. Gossage; Charles A. Greer; Trese Leinders-Zufall; C. Geoffrey Woods; John N. Wood; Frank Zufall
Journal:   Nature
Volume:   472
Issue:   7342
Year:   2011
Pages:   186
DOI:   10.1038/nature09975
Publication date:   23-Mar-2011
Facts, background information, dossiers
More about Nature Publishing Group
Your browser is not current. Microsoft Internet Explorer 6.0 does not support some functions on Chemie.DE